Lise Bjørkhaug Gundersen
Field of work
I am a molecular biologist at my core, researching to understand the molecular pathological relationship between genetics and the development of hereditary diabetes (Maturity-onset diabetes of the young: MODY1, MODY3, and MODY5). I also research changes in biological markers due to exercise/sports.I teach medical laboratory technology in the bachelor's and master's programs of biomedical engineering, where I am the program director for the MAMLT master's program. I use research-based cases in my teaching so that students can gain a better understanding of the relationship between cause and disease for personalized treatment of MODYs. I involve bachelor's, master's, and PhD students in research projects, where the issue may be understanding the effect of gene damage on the development of hereditary diabetes, or the effect of biological markers on performance in sports (interdisciplinary and cross-faculty projects).
Courses taught
- Medical laboratory tehcnology (BSc)
- Advanced medical laboratory analyses (MSc)
Research areas
- Inherited (monogenic) diabetes and genetics
- Type 2 diabetes and genetics
- Health and performance in sports
Research groups
- BIO304, Medical laboratory technology IV, Fall 2025
- BIO350, Bachelor Thesis - Biomedical laboratory sciences, Spring 2026
- BIO502, Advanced methodology in medical laboratory science, Spring 2026
- BIO599, Master Thesis in Medical Laboratory Science, Spring 2026
- BIO599, Master Thesis in Medical Laboratory Science, Fall 2025
Publications
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Acute response in circulating microRNAs following a single bout of short-sprint and heavy strength training in well-trained cyclists
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Functional characterization of HNF4A gene variants identify promoter and cell line specific transactivation effects
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Characterisation of HNF1A variants in paediatric diabetes in Norway using functional and clinical investigations to unmask phenotype and monogenic diabetes
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A novel splice-affecting HNF1A variant with large population impact on diabetes in Greenland
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The contribution of functional HNF1A variants and polygenic susceptibility to risk of type 2 diabetes in ancestrally diverse populations